Answer:
Cancer cells achieve proliferative immortality by activating or upregulating the normally silent human TERT gene (hTERT) that encodes telomerase, a protein with reverse transcriptase activity that complexes with other proteins and a functional RNA (encoded by hTR, also called hTERC) to make a ribonucleoprotein enzyme.
Explanation:
A rare cell that escapes crisis almost universally does so by reactivating telomerase and this cell can now become a cancer cell with limitless potential to divide. Almost all cancer cells have short telomeres and thus inhibitors of telomerase should drive such cancer cells into apoptotic cell death. Yet, each time a cell divides, the telomeres get shorter. When they get too short, the cell no longer can divide and becomes inactive or "senescent" or dies. This process is associated with aging, cancer, and a higher risk of death.
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D ribosomes because it makes the energy for the cells
Answer:
Proten W
Explanation:
SDS-PAGE gel is a method used for the separation of proteins in which proteins are separated based on their length (smaller proteins move faster through the gel, due to less resistance).
When treated with proteolytic enzymes, proteins are cut and become short fragment. This means that the fragments formed after the use of proteolytic enzymes, will move faster and thus, migrate a longer distance. Proteolytic enzymes in Set 2 cells will act only on plasma membrane proteins (because they cannot penetrate), while in Set 2 they will act on both, plasma membrane and interior proteins. Control group will have only the large fragments (not treated with enzyme).
Protein W travels the same distance on a gel of proteins from Set 1 and Set 2, but different than control group. It means that the proteolytic enzyme worked the same on Set 1 and Set 2.
C. The exact amount of nonrenewable energy resources is not currently known.
