Ureter, the structure that carries urine from the kidney to the bladder is called Ureter
Answer:
1. The difference between the normal hemoglobin protein DNA sequence and the sickle cell hemoglobin DNA sequence is a base to base shift, in this case adenine (GAG) to thymine (GTG).
2. The difference affects the amino acid sequence of the protein by replacing glutamic acid (Glu) with valine (Val).
Explanation:
In sickle cell anemia, a change in the DNA nucleotide sequence is observed, where adenine is substituted by thymine, whose expression is the change in the amino acid sequence of globine β, incorporating valine instead of glutamic acid. This represents a molecular mutation - point mutation - by subtitution, which corresponds to missense mutation.
<u>Normal hemoglobin protein in a RBC</u>
DNA CTG ACT CCT GAG GAG AAG TCT
Amino acids Leu Thr Pro Glu Glu Lys Ser
<u>Sickle cell hemoglobin protein in a RBC</u>
DNA CTG ACT CCT <em>GTG</em> GAG AAG TCT
Amino acids Leu Thr Pro <em>Val</em> Glu Lys Ser
When GAG is transcribed to mRNA, the CUC codon is obtained, which codes for glutamic acid. Thymine substitution causes the DNA sequence to change to GTG, which is transcribed as CAC, the codon that encodes the amino acid valine. The <u>change from glutamic acid to valine in β-globin causes an altered hemoglobin, giving the abnormal erythrocytes observed in sickle cell disease</u>.
Youre using the distributive property. sk you multiply negative three by x and negative 5. itt equals negative three x and positive 15. you subtract 15 frkm 45. which leaves 30. then you divide by negative three. the answer is negative ten.
Answer:
This question lacks options, options are: Mr. M's cells are depolarizing too easily. Blocking Nat channels will make it harder for them to depolarize, bringing their sensitivity back to normal O Mr. M has too little K* leaving his cells, so we need to block the Nat channels so the Kt channels can stay open and his cells can repolarize properly. Mr. M has too little K* leaving his cells, so we need to block the Nat channels so the Na /K+ ATPase can move Kt out of the cell effectively O Blocking Na* channels cannot help- it will only make his cells fire less, and he already has a weak heartbeat. Mr. M's cells are not repolarizing properly. Blocking Nat channels will help them repolarize normally, bringing their resting potential back to normal.
The correct answer is ''Mr. M's cells are depolarizing too easily. Blocking Na+ channels will make it harder for them to depolarize, bringing their sensitivity back to normal.''
Explanation:
Calcium administration is an emerging treatment modality aimed at restoring the transmembrane electrical gradient of cardiac myocytes. It probably achieves this goal by reducing the resting membrane potential of cells. Calcium antagonizes the effect of hyperkalemia on cardiac conduction, that is, it antagonizes the effects of K on the heart. Its onset of action is immediate, in a few minutes. Calcium gluconate antagonizes the excitability of the cardiac membrane, that is, it decreases the excitability of the membrane and it does not affect serum levels, it is generally accepted that calcium should be administered when there are ECG changes associated with hyperkalemia.
Answer:
Nitrification.
Explanation:
It is a part of the Nitrogen Cycle. The bacterias can convert ammonia into nitrite and nitrate, these compounds are fundamentally important for all life. They are used for the creation of proteins and amino acids.
I hope this answer helps you.
