Answer:
The options
A) "Medications are needed to prevent heart failure."
B) "I will take aspirin, atenolol, and captopril indefinitely."
C) "My blood pressure should be less than 130/80 mm Hg."
D) "Daily estrogen will prevent another heart attack."
The CORRECT ANSWER IS D)
D) "Daily estrogen will prevent another heart attack."
Explanation:
A reduction in the natural hormone estrogen levels could be a causative factor in heart disease increase within post-menopausal women. Estrogen is thought to show a positive impact on the inner layer of artery wall, functioning in such a way to keep blood vessels elastic.
In spite of the advantages of estrogen, the American Heart Association speaks against employing postmenopausal hormone therapy to decrease the likelihood of coronary heart disease or stroke as some research works have revealed that it vividly do not decrase the risk of heart attack but increases the likelihood of blood clots and stroke.
1. k
2. d
3. c
4. j
5. f
6. e
7. l
8. h
9. g
10. b
11. i
12. a
I believe these are all right correct me if I am wrong
Answer: 2. Precipitation
Explanation:
Aerobic cellular respiration requores oxygen. Photosynthesis does, as well.
Answer:
Hepato- and nephrotoxicity of fluoride have been demonstrated in animals, but few studies have examined potential effects in humans. This population-based study examines the relationship between chronic low-level fluoride exposure and kidney and liver function among United States (U.S.) adolescents. This study aimed to evaluate whether greater fluoride exposure is associated with altered kidney and liver parameters among U.S. youth.
This cross-sectional study utilized data from the National Health and Nutrition Examination Survey (2013–2016). We analyzed data from 1983 and 1742 adolescents who had plasma and water fluoride measures respectively and did not have kidney disease. Fluoride was measured in plasma and household tap water. Kidney parameters included estimated glomerular filtration rate (calculated by the original Schwartz formula), serum uric acid, and the urinary albumin to creatinine ratio. Liver parameters were assessed in serum and included alanine aminotransferase, aspartate aminotransferase, alkaline phosphatase, blood urea nitrogen, gamma-glutamyl transferase, and albumin. Survey-weighted linear regression examined relationships between fluoride exposure and kidney and liver parameters after covariate adjustment. A Holm-Bonferroni correction accounted for multiple comparisons.
The average age of adolescents was 15.4 years. Median water and plasma fluoride concentrations were 0.48 mg/L and 0.33 μmol/L respectively. A 1 μmol/L increase in plasma fluoride was associated with a 10.36 mL/min/1.73 m2 lower estimated glomerular filtration rate (95% CI: −17.50, −3.22; p = 0.05), a 0.29 mg/dL higher serum uric acid concentration (95% CI: 0.09, 0.50; p = 0.05), and a 1.29 mg/dL lower blood urea nitrogen concentration (95%CI: −1.87, −0.70; p < 0.001). A 1 mg/L increase in water fluoride was associated with a 0.93 mg/dL lower blood urea nitrogen concentration (95% CI: −1.44, −0.42; p = 0.007).
Fluoride exposure may contribute to complex changes in kidney and liver related parameters among U.S. adolescents. As the study is cross-sectional, reverse causality cannot be ruled out; therefore, altered kidney and/or liver function may impact bodily fluoride absorption and metabolic processes.
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