Shortness of breath or difficulty in breathing is also called dyspnea and can be acute or chronic. It has various causes, but mainly can be caused by a problem in the heart or the lungs. Since your heart and lungs are both involved in the transportation of the oxygen to the tissues and the removal of carbon dioxide, any problems occurring to these systems can affect breathing.
B-type Natriuretic Peptide (BNP) reflect the systolic and diastolic activity of the heart and its blood levels can show any heart failure. A BNP test and can help the nurse decide whether the cause of the dyspnea is a heart failure or some respiratory problem.
A particular triplet of bases in the template strand of DNA is 5' agt 3'. the corresponding codon for the mRNA transcribed is<u> 3' UCA 5'</u>
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It serves as a link between DNA's genetic code and proteins' amino acid sequences.
The codons in the messenger RNA (mRNA) are complementary to the nucleotide sequence on the DNA template strand.
Then it controls the synthesis of amino acids with the aid of tRNA and ribosomes.
Its name, messenger RNA, refers to the fact that it carries genetic information. The single strands of adenine, cytosine, guanine, and uracil that make up the mRNA molecule are held together by a sugar phosphate backbone.
Two codons: His, Lys, Phe, Tyr, Asn, Asp, Cys, Gln, Glu, Codons Ile, STOP, and three ("nonsense"). Ala, Gly, Pro, Thr, and Val are the four codons. None of the five codons.
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Answer:
Due to number of amino acids.
Explanation:
The main differences might be expected in the amino acid composition of X versus Y because the X polypeptide is made up of one smaller unit of amino acid while on the other hand, Y polypeptide is made up of four smaller units of amino acids. The name of X is monomer which means composed of one amino acids while Y exists as a tetramer which means it has four amino acids. So we can conclude that the main difference between X and Y is the presence of number of amino acids.
Answer:
The short answers are Yes, it's random, and Yes, it "waits" for some time.
Different tRNA's just float around in the cytoplasma, and diffuse more or less freely around. When one happens to bump into the ribosome, at the right spot, right orientation, and of course which has an anticodon matching the codon in frame of the mRNA being translated, it gets bound and takes part in the synthesis step that adds the amino acid to the protein that is being synthesized.
The concentration of the various species of tRNA is such that translation occurs in a steady fashion, but there is always some waiting involved for a suitable tRNA to be bound. In that waiting time, the ribosome and mRNA stay aligned - that's because the energy that is required to move the to the next position is delivered as part of the same chemical reaction that transfers the amino acid from the tRNA to the protein that is being synthesized.
I'm not entirely sure what happens if there is significant depletion of a particular species of tRNA, but I think it's likely the ribosome / RNA complex can disassemble spontaneously. But spontaneous disassembly can't be something that occurs very easily after translation was initiated, since we would end up with lots of partial proteins which I expect would be lethal very soon.
(Can't know for sure though, but it would be very hard to set up an experiment to measure just what will happen and even if you got a measurement it would be hard to figure out how it applies to normal, living cells. I can't imagine tRNA depletion occurs in normal, healthy living cells.)