Klinefelter's syndrome is a result of chromosomal nondisjunction mutations.
<h3>What is Klinefelter's syndrome?</h3>
Klinefelter's syndrome is a result of the presence of an extra X chromosome in germinal cells.
In genetics, this type of mutation is referred to as chromosomal nondisjunction (aneuploidies).
In conclusion, Klinefelter's syndrome is due to chromosomal nondisjunction.
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The type of muscle found at the heart is the cardiac muscle tissue. This tissue has the phyical striped characteristic of a skeletal muscle tissue and works like a involuntary muscle because it works without our control
This type of tissue is packed wirh mitochondria which provides it with alot of energy. This is very crucial becaise the heart has to constantly beat
Unsaturated fatty acids are a component of the phospholipids in cell membranes and help maintain membrane fluidity. The Phospholipids contain a variety of unsaturated fatty acids, when compressed, the “kinks” in their tails push adjacent phospholipid molecules away, that helps in maintain fluidity in the membrane. Unsaturated fatty acids have at least one double bond, creating a "kink" in the chain, the absence of double bonds decreases fluidity, making the membrane very strong and stacked tightly.
The ratio of saturated and unsaturated fatty acids determines the fluidity in the membrane at a temperature, at appropriate temperatures the phospholipids have enough kinetic energy to overcome the intermolecular forces holding the membrane together, which increases membrane fluidity.
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Ricin comes under the category of functionally related toxins, in a combination known as RIPs (ribosome inactivating proteins). This disables ribosomes and stops the process of protein synthesis.
They directly associate with and inactivate the ribosomes or modify the factors taking part in the process of translation, generally the elongation step. These proteins bring about depurination of adenine at position 4324 in the 28 S rRNA.
This further inhibits the generation of a critical-stem-loop configuration to which the elongation factor is considered to combine at the time of the translocation step of translation. The ultimate outcome of this activity is the complete inhibition of cellular translation.
