Answer:
The <em>brain stem</em> is a bundle of nervous tissue at the base of the brain that connects the cerebrum to the spinal cord and sends messages between different parts of the body and the brain
Explanation:
Answer:
a. No, it is not possible.
A heterozygous female carries one copy of functional gene which is enough for the production of clotting factor. Hemophilia does not show continuous variation or polygenic inheritance and thus, its level does not depend on the number of normal alleles.
It that was the case, then all males would show hemophilia in some parts of the body as they only carry one X chromosome and thus, only one functional gene.
Thus, heterozygotes are only the carriers of the disease, they do not show any symptom of the disease.
b. In perspective of homozygosity or heterozygosity, the rate of blood clotting should be the same as both of them have functional gene. As mentioned above, it does not show continuous variation so, it will not show any increased or decreased rate of clotting in homozygotes or heterozygotes.
However, in reality, the rate of clotting depends on the concentration of clotting factor present in blood plasma. This percentage depends on the physiology of a person but not on the number of alleles present. For example, proteins or enzymes required for gene expression, et cetera.
Answer:
That foxes, wolves, and Dingoes have a common ancestor they all share but, however each species branches off because of evolution.
Oncogenes are most like the gas pedal on a car, while tumor suppressor genes are most like the brakes on a car. Oncogenes and tumor suppressor genes are mutated in cancer cells.
Oncogenes are genes overexpressed in cells in which they should not be expressed, thereby leading to cancer.
Some examples of oncogenes are growth factors such as, for example, the Platelet-derived growth factor (PDGF) or Epidermal growth factor (EGF).
Conversely, tumor suppressor genes are genes that act to regulate cell division and replication, thereby their inactivation also leads to cancer.
A well-known example of a tumor suppressor gene is the p53 gene that acts to control cell division and apoptosis (programmed cell death).
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