Answer:
Aspirin works by inhibiting the production of prostaglandins. Aspirin inhibits the formation of prostaglandins by combining with the COX enzymes. Prostaglandins function as messenger molecules to monitor different physiological procedures in distinct regions of the body. One of the prime activities of prostaglandins is to stimulate inflammation and pain.
Prostaglandins are also the essential controller of platelet aggregation. By changing the COX enzymes inside the platelets, aspirin makes platelets to lose the stickiness, which is required to instigate clotting of blood.
There are two forms of cyclooxygenase, that is, COX-1 and COX-2. COX-1 generates prostaglandins and COX-2 mediates pain and swelling in response to tissue injury. Aspirin prevents both COX-1 and COX-2 functioning, while COX-2 is the therapeutic target of the drug.
However, it is the association of aspirin with COX-1 in the gastrointestinal tract, which results in the unwanted side effects of the drug. COX-1 is required to sustain a thick lining of the stomach. As aspirin inhibits the COX-1 enzyme, thus, the continuous use of the drug can result in the thinning of mucus, which safeguards the stomach from gastric juices.
In such cases, stomach bleeding, ulcers, and in certain situations perforation of the stomach can take place. Therefore, aspirin exhibits both bad and good effects.
Answer:Inducible;inducer
Explanation:
Induction is the phenomenon of increased synthesis of protein or and enzyme in response to certain signal. Such enzymes are called inducible and the signals are called inducer. Induction is turning ''on''the switch of a gene.
When M is present in the medium, M binds the repressor protein. The repressor-M complex is inactive which does not bind to the operator region of LM. Now RNAP can transcribe the structural genes of LM.
M switches the gene ''on''. Hence M is an inducer of LM. LM is an inducible operon
Wax can com from a candle and that is a solid so A and also D mixtures
