D) #recombinant=116+601+4+2+113+625=1462
#nonrecombinant=2538+2708=5246
RF=R/(N+R)=1462/6708=0.2
E) I=1-Q
I-interference
Q-coefficient of coincidence
Q=O2Xo/E2xo(2xo-double crossovers)
O2xo=6
P=(distance from C1 to Sh/100)*(distance from Sh to Wx/100)
=(3.38/100)*(18.28/100)=0.006
E2xo=0.006*6708=40.248
Q=6/40.248=0.15
I=1-0.15=0.85
Answer:
No, telomerase is not an oncogene. It prevents the senescence that would occur due to shortened telomeres, but the cell proliferation might still be mitogen-dependent.
Explanation
telomerase is not responsible for causing deregulation while oncogenes cause deregulation .
Telomeres length shorten after the cell division which stops them to divide again and cell die.
Telomerase prevents this decline in some kinds of cells, including stem cells, by lengthening telomeres, and the hope was that activating the enzyme could prevent senescence.
The fossil shows evidence of how animals lived on Earth in the past and shows history about animals that may be extinct now or have evolved since then, which would show development in the animal in the fossil.
Cerebellum smoothes and coordinates the movement of skeletal muscle.
