Answer:
ATP, Mitochondria, Cellular respiration.
Explanation:
The Krebs cycle of, cellular respiration, in the mitochondria results to the an accumulation of protons in the inter-membrane space of the organelle. This proton motive force is harnessed in the electron transport chain by ATP synthase to generate ATPs.
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The correct answer is option A, that is, placer deposits.
The concentration of heavy deposits known as the placer deposits are produced by the gravitational action, generally found in streams. The minerals that take place as placer deposits comprise copper, gold, magnetite, cassiterite, and copper.
The formation of placer deposits takes place when the metal-bearing rock on land is weathered and the generated debris is washed down to the sea by the rivers. Further by the action of waves, currents, and rides the particles of heavy metal gets amassed to produce mineral sand deposits. These generally occur as beach deposits.
Answer:
The protein is targeted to Golgi apparatus.
Explanation:
Protein targeting or sorting is a process by which synthesized proteins are transported to their appropriate localizations in the cell or outside it. Proteins can be targeted to the inner space of an organelle (such as Golgi apparatus or endoplasmic reticulum) or its membrane, plasma membrane, or to exterior of the cell via secretion.
Usually, protein contains signal sequence on the N terminus that is involved in destination targeting.
Answer:
C) Through genomic imprinting, methylation regulates expression of the paternal copy of the gene in the brain.
Explanation:
The pattern of gene expression wherein either paternal or maternal gene is expressed in specific cells while the other one is prevented from expression is known as genomic imprinting.
In the given example, the maternal copy of the gene on chromosome 15 is expressed in brain cells while its paternal copy is not expressed in these cells. Hence, the pattern of expression of this gene is regulated through genome imprinting. One of the mechanism is methylation of cytidine residues of CpG islands of the DNA that are more frequently present within promoters of the genes.
When the cytidine residues of these sequences are methylated into 5-methylcytidine, the transcription factors do not bind to these promoters preventing the expression of these genes.
Hence, methylation of cytidine residue in CpG islands of the promoters of the gene present on chromosome 15 could have silenced its expression in brain cells.
