Answer and explanation :
there are many problem due to lack of pneumotaxic and stetch receptor neurons on ventilation
DUE TO LACK OF PNEUMOTAXIC CENTER :
- pneumotaxic center is present in the nucleus which is used to transmit impulse to the inspiratory area which is not possible due to lack of pneumotaxic center
- it controls the duration of filling of lungs
- it also control the medullary respiratory system which is not possible due to lack of this
DUE TO LACK OF STRETCH RECEPTOR :
- The hearing breuer inflation reflex is generated by stretch receptor neuron which is not generated due to lack of stretch receptor
- when stretched these receptors send the inhibitory impulses through the vegus nerve to the DMG neuron which is not possible due to lack of this
Risks of deep vein thrombosis (DVT) and pulmonary embolism (PE) in chronic pancreatitis (CP) are <u>unclear</u>.
The CP cohort showed a 2.95-fold greater adjusted hazard ratio (aHR) for DVT and a 4.51-fold greater aHR for PE than the non-CP cohort. Substantial risks of DVT and PE were evident in patients with CP aged < 55 years.
The CP cohort with comorbidities showed increased risks of DVT and PE as compared with the non-CP cohort.
Hence the risks of DVT and PE are significantly higher in CP patients than in the general population.
To learn more about Comorbid risks of deep vein thrombosis and pulmonary thromboembolism, here
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The consequences if presynaptic action potentials In an axon release insufficient acetylcholine to depolarize a skeletal muscle fiber to threshold
Explanation:
When an action potential reaches a neuromuscular junction, it causes acetylcholine to be released into this synapse. The acetylcholine binds to the nicotinic receptors concentrated on the motor end plate, a specialized area of the muscle fibre's post-synaptic membrane.
Acetylcholine is the neurotransmitter used at the neuromuscular junction—in other words, it is the chemical that motor neurons of the nervous system release in order to activate muscles. ... In the brain, acetylcholine functions as a neurotransmitter and as a neuromodulator.
Answer:
Explanation: glutamate and glycine (or D-serine) bind to it, and when activated it allows positively charged ions to flow through the cell membrane. The NMDA receptor is very important for controlling synaptic plasticity and memory function. The NMDAR is a specific type of ionotropic glutamate receptor.
