Answer:
8 kg
Explanation:
Yeah. 8kg of chocolate! Seems so funny, but that is the answer for this problem.
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Please see the step-by-step solution for this problem in the picture attached below.
Hope this can help you. Have a nice day!
# 1. Carbohydrates
# 2. Lipids
# 3. Proteins and Amino Acids
# 4. Isoprenoids and Pigments
Answer:
Genotype ratio: 1 (RR) : 2 (RW) : 1 (WW)
Phenotype ratio: 1 Red : 2 Roan : 1 White
Explanation:
Codominance is a kind of inheritance pattern in which both alleles are equally dominant in a gene. This means that both alleles when in a heterozygous state will be simultaneously expressed. According to this question, allele for a red coat (R) and the allele for a white coat (W) results in offspring with a roan coat (RW) in cattle.
Hence, in a cross between a a roan cow (RW) and a roan bull (RW), the following gametes will be produced: R and W.
Using these gametes in a punnet square (see attached image), the offsprings with the following genotypes will be produced: RR, RW, RW, WW.
Therefore, based on the results of the cross;
Genotype ratio: 1 (RR) : 2 (RW) : 1 (WW)
Phenotype ratio: 1 Red : 2 Roan : 1 White
Answer:
Anchoveta feed on large zooplankton whereas sardines feed mainly on phytoplankton. Use this information to suggest why sardine numbers are less
likely to collapse during an El Niño event?
Reason why sardine are less likely to collapse during an El Nino event is as a result of sardine survival is neither dependent on zooplankton nor anchoveta rather sardine is dependent on phytoplankton which is not scarce
Explanation:
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
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