Conversion of 1 mol of acetyl-CoA to 2 mol of Co2 and CoA via the citric acid cycle results in the net production of 12 mol of ATP.
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What is acetyl-coA?</h3>
Acetyl-CoA is produced in the mitochondrial matrix by oxidative decarboxylation of pyruvate from glycolysis, oxidation of long-chain fatty acids, or oxidative degradation of specific amino acids. The acetyl-CoA is then oxidized for energy production in the TCA cycle.
Mitochondrial enzymes are used in the citric acid cycle. The acetyl group of acetyl-CoA is fused with oxaloacetate in the first phase, which is mediated by citrate synthase. Citrate is formed as a result of the release of CoA-SH and heat. Dehydration and rehydration convert citrate to isocitrate.
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Answer: Mutations in the GALT, GALK1, and GALE genes cause galactosemia.
Explanation: Galactosemia is a disorder that affects how the body processes a simple sugar called galactose. A small amount of galactose is present in many foods. It is primarily part of a larger sugar called lactose, which is found in all dairy products and many baby formulas. The signs and symptoms of galactosemia result from an inability to use galactose to produce energy.Classic galactosemia, also known as type I, is the most common and most severe form of the condition. If infants with classic galactosemia are not treated promptly with a low-galactose diet, life-threatening complications appear within a few days after birth. Affected infants typically develop feeding difficulties, a lack of energy (lethargy), a failure to gain weight and grow as expected (failure to thrive), yellowing of the skin and whites of the eyes (jaundice), liver damage, and abnormal bleeding. Other serious complications of this condition can include overwhelming bacterial infections (sepsis) and shock. Affected children are also at increased risk of delayed development, clouding of the lens of the eye (cataract), speech difficulties, and intellectual disability. Females with classic galactosemia may develop reproductive problems caused by an early loss of function of the ovaries (premature ovarian insufficiency). Galactosemia type II (also called galactokinase deficiency) and type III (also called galactose epimerase deficiency) cause different patterns of signs and symptoms. Galactosemia type II causes fewer medical problems than the classic type. Affected infants develop cataracts but otherwise experience few long-term complications. The signs and symptoms of galactosemia type III vary from mild to severe and can include cataracts, delayed growth and development, intellectual disability, liver disease, and kidney problems.
Answer:
Muscle Build and Building a tolerance
Explanation:
As you work out or even just moving excessively, your muscles tear in tiny amounts to heal the body sends cell to patch up that area over time the cells will just keep stacking up. This is where the tolerance comes in since their is so many cells built up its harder for them to "rip" It sounds like a huge rip but its more of a tiny little rip not even enough to hurt.(until the next day.)
Hypotension is a sign of potential hypovolemia.
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What is Hypotension?</h3>
- When your blood pressure is substantially lower than you would expect, you have hypotension, or low blood pressure. It can occur as a standalone disorder or as a sign of a variety of other conditions.
- Although it might not produce symptoms, if it develops, it could need to be treated by a doctor. Total hypotension Your blood pressure is lower than 90/60 mmHg at rest (millimeters of mercury).
- When you get up from a sitting position, you experience orthostatic hypotension, which causes your blood pressure to plummet within three minutes.
- Your systolic (top) pressure must drop by at least 20 mmHg, and your diastolic (bottom) pressure must drop by at least 10 mmHg. Postural hypotension is another name for this since it occurs when a person's posture shifts.
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