Saturated zone
The correct answer is saturated zone. This area contains permeable rocks and soil here is totally filled with water or we can say is saturated with the water. This zone is called as saturated zone.
The most appropriate answer would be Winter !!
As in southermost past there is only two season winter and summer !! So as the earth is moving away from sun and thus the southern part will be far away and northern part will be facing sun !! So C !!
Answer:
The autonomic nervous system is in charge of controlling visceral effectors. Traditionally, it is described by its peripheral nervous components (ganglia, nerves and plexuses) and two divisions are distinguished: the sympathetic and the parasympathetic. Transmission of the excitatory stimulus through the synaptic cleft occurs by release of neurotransmitters; the neurotransmitters of the sympathetic and parasympathetic nervous system are mainly norepinephrine (NA) and acetylcholine (AC). The NA-secreting fibers are called adrenergic and those that secrete AC, cholinergic. All preganglionic neurons, both those of the sympathetic nervous system and those of the parasympathetic nervous system, are cholinergic. The neuron that releases the neurotransmitter is called a presynaptic neuron. The signal receptor neuron is called a postsynaptic neuron. Depending on the type of neurotransmitter released, postsynaptic neurons are either stimulated (excited) or de-stimulated (inhibited).
Explanation:
The autonomic nervous system is the part of the central and peripheral nervous system that is responsible for the regulation of the involuntary functions of the organism, the maintenance of internal homeostasis and the adaptive responses to variations in the external and internal environment and two divisions are distinguished: the sympathetic and the parasympathetic. Acetylcholine is the preganglionic neurotransmitter of both divisions of the S.N.A. (sympathetic and parasympathetic) and also of the postganglionic neurons of the parasympathetic. The nerves at whose endings acetylcholine are released are called cholinergic. Norepinephrine is the neurotransmitter of postganglionic sympathetic neurons. The nerves into which norepinephrine is released are called adrenergic. Within the efferent sympathetic impulses, the postganglionic neurons that innervate the eccrine sweat glands and some blood vessels that supply the skeletal muscles are of the cholinergic type. Both acetylcholine and norepinephrine act on the different organs to produce the corresponding parasympathetic or sympathetic effects. The peripheral nerve endings of the sympathetic form a reticulum or plexus from which the terminal fibers come in contact with the effector cells. All the norepinephrine in peripheral tissues is found in the sympathetic endings in which it accumulates in subcellular particles analogous to the chromaffin granulations of the adrenal medulla. The release of norepinephrine at nerve endings occurs in response to action potentials that travel through nerve endings. The receptor, when stimulated by catecholamines, sets in motion a series of membrane changes that are followed by a cascade of intracellular phenomena that culminate in a measurable response. There are two classes of adrenergic receptors known as alpha and beta. These two classes are again subdivided into others that have different functions and that can be stimulated or blocked separately. Norepinephrine primarily excites alpha receptors and beta receptors to a small extent. The neurotransmitter acetylcholine is synthesized at the axonal terminal and deposited in synaptic vesicles. Acetylcholine activates two different types of receptors, called muscarinic and nicotinic receptors. Acetylcholine (AC) synthesis takes place at presynaptic termination by acetylation of choline with acetyl-coenzyme A, a reaction catalyzed by acetylcholinetransferase. The energy required for the release of a neurotransmitter is generated in the mitochondria of the presynaptic terminal. Binding of neurotransmitters to postsynaptic membrane receptors produces changes in membrane permeability. The nature of the neurotransmitter and the receptor molecule determines whether the effect produced will be one of excitation or inhibition of the postsynaptic neuron.
After first exposure to an antigen, it can take about three weeks for antibodies to reach a detectable level. The body will react to disease by creating antibodies and testing their effectiveness against the unknown antigen.
Antibodies are the body's way of fighting off foreign threats. Also known as immunoglobulin, they detect and attack pathogenic bacteria and viruses. These antibodies detect a protein that is unique to the surface of the pathogen called the antigen.
At times, we can see an increase in antibodies for one disease, for example, <em>Lyme disease</em>, in the presence of <em><u>non-Lyme disease antigens.</u></em> This is often due to antigenic variation, which is a method used by pathogens to mask their respective antigens. There are also general use antibodies that will increase in reaction to any pathogen.
As with any illness, a patient who receives treatment sooner will fare better than those whose treatment is delayed. This is due to the fact that earlier treatment with antibiotics will allow the body to fight off the infection before the bacteria can reproduce further.
After the first immune response, antigen-specific antibodies will remain for some time to provide the body with "immunity" to the pathogen, while <u>general-purpose antibodies will return to a base value. </u>
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