I think the answer is most likely be J.
The first (F) one the population of the predator increases hugely while the population of the prey was neutral. And so both population didn’t seem to have any connection. Same goes for H. Graph G doesn’t make sense at all the population of the prey didn’t exist throughout the time in the graph but only exist in one single point of time and then just vanish again so that shouldn’t be the answer either.
In graph J, you can see the correlation between the two populations as the predator goes up and so does the prey.
You can search up on google predator-prey relationship graph to get better understanding.
Answer:
Compounds from different organisms are being used widely bu humans for their benefits. Some of the compounds are used as drugs. For example, penicillin is used as a drug or antibiotic. There are many plants which provides humans with other types of medicines and herbal remedies. There are other compounds which are used for cancer treatment. For example, taxol is a compound obtained from trees which are being used as anticancer drugs.
Explanation:
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Answer:
For the benefits we have had improvements in communication and transportation which has caused an increase in economic growth.
For the disadvantages this causes (urbanization) will allows people to live in urban areas.
Explanation:
Answer:
The order must be K2→K1, since the permanently active K1 allele (K1a) is able to propagate the signal onward even when its upstream activator K2 is inactive (K2i). The reverse order would have resulted in a failure to signal (K1a→K2i), since the permanently active K1a kinase would be attempting to activate a dead K2i kinase.
Explanation:
- You characterize a double mutant cell that contains K2 with type I mutation and K1 with type II
mutation.
- You observe that the response is seen even when no extracellular signal is provided.
- In the normal pathway, i f K1 activat es K2, we expect t his combinat ion of two m utants to show no response with or without ext racell ular signal. This is because no matt er how active K1 i s, it would be unable to act ivate a mutant K2 that i s an activit y defi cient. If we reverse the order, K2 activating K1, the above observati on is valid. Therefore, in the normal signaling pathway, K2 activates K1.
