Answer:
Makali may produce only small amounts of a non-mutated (wild-type) GALT enzyme.
Makali may have normal amounts of GALT, but the enzyme may be mutated.
Explanation:
Makali is lactose intolerant because of his ancestry. Because of this he is not able to digest any lactose which indirectly protected him from galactosemia. Thus he must avoid consuming galactose. He has a low GALT or galactose 1‑phosphate uridylyltransferase activity. He has a normal amount of GALT and may produce only small amounts.
Ionizing radiation, for example X-rays, gamma rays and alpha particles
Answer:
An acid is a substance or compound that releases hydrogen ions (H+) when in solution. In a strong acid, such as hydrochloric acid (HCl), all hydrogen ions (H+), and chloride ions (Cl-) dissociate (separate) when placed in water and these ions are no longer held together by ionic bonding.
The origin of congenital adrenal hyperplasia occurs when circulating cortisol levels are low.
Congenital adrenal hyperplasia (CAH) is an autosomal recessive disease which results from mutations of genes for enzymes involved in the biochemical steps of production of mineralocorticoids, glucocorticoids or sex steroids from cholesterol by the adrenal glands (steroidogenesis). <span>Low cortisol production results in rising levels of ACTH (adrenocorticotropic hormone) because cortisol usually inhibits ACTH production. This increased ACTH production induces overgrowth (hyperplasia) and overactivity of the steroid-producing cells of the adrenal cortex.</span>
