Answer:
Angiotensin-converting enzyme (ACE) inhibitors are commonly prescribed to treat high blood pressure, heart problems and other conditions. Find out how they work and their possible side effects.
Angiotensin-converting enzyme (ACE) inhibitors help relax veins and arteries to reduce blood pressure. ACE inhibitors prevent an enzyme in your body from producing angiotensin II, a substance that narrows your blood vessels. This narrowing can cause high blood pressure and force the heart to work harder. Angiotensin II also releases hormones that raise blood pressure.
In addition to high blood pressure, angiotensin-converting enzyme inhibitors prevent, treat or improve symptoms in conditions such as the following:
Coronary artery disease
Heart failure
Diabetes
Certain chronic kidney diseases
Heart attacks
Scleroderma: a disease that involves hardening of the skin and connective tissues
Migraines
The doctor may prescribe other medications in addition to an angiotensin-converting enzyme inhibitor, such as a diuretic or a calcium antagonist. Angiotensin-converting enzyme inhibitors should not be taken together with angiotensin receptor blockers or with direct renin inhibitors.
Angiotensin-converting enzyme inhibitors work better for younger people than for older people. They also work better for white people than for black people. The doctor may recommend a different medication.
The amount of deer moving about in urban areas would increase. Instead of being in their natural habitat, deer would be walking around, grazing in the yards of humans to try and find food. This may also lead to an increase in wildlife related collisions, as more deer would be trying to cross busy roads to find more to eat.
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1. Explain why neither cyclins nor kinases alone can cause a cell to progress through the cell cycle.
As cyclin accumulates, it activates their kinases that turn on the pathway to mitotic spindle formation, and so on.
2. How do controls of the cell cycle protect multi-cellular organisms from accumulating large numbers of damaged or defective cells?
The checkpoint control is responsible for multi-cellular organisms for not accumulating large numbers of damaged or defective cells. Checkpoint controls consist of proteins that detect mistakes and damage and quickly halt the cell cycle until repairs are made. When this occurs, the cell is said to be in cell-cycle <span>arrest.
</span>3. What is the difference between a cancerous tumor and metastasis?
Cancer is cause by mutations in the genes that encode these proteins can lead to uncontrolled growth. Cancer is when there is uncontrolled cell growth and reproduction. Metastasis is caused by tumors when they grow and interfere with the surrounding tissue or cells and break off and spread around the body. Cancerous tumors cause metastasis, and tumors are caused by mutations in genes that lead to uncontrolled growth.
4. What are the functions of tumor-suppressor genes and protoncogenes in noncancerous cells?
The genes that encode the checkpoint proteins are called tumor suppressors because they suppress the development of cells into tumors. If mutations inactivate these genes, the cell-cycle break is removed with or without a signal from the outside. Proto-oncogene’s are involved in promoting cell division, mutations can cause them to become oncogenes, or cancer genes which stimulate cells to leave G0 and divide whether or not it is a signal.
Answer:
NAD is a coenzyme found in all cells. It consists of two nucleotides linked through their phosphate groups with a nucleotide that contains an adenosine ring and another that contains nicotinamide.
Explanation:
In metabolism, NAD participates in oxidation reduction reactions. This coenzyme is found in two forms in the cells: NAD and NADH. NAD accepts electrons from other molecules and is reduced, forming NADH, which you can use as a reducing agent to donate electrons. These electron transfer reactions are the main function of the NAD.
In living organisms, NAD can be synthesized from scratch from the amino acids tryptophan or aspartic acid. Some NADs are found in nicotinamide adenine dinucleotide phosphate (NADP), whose chemistry is similar to NAD, although it has different functions in metabolism.
The answer to this would be “ T-A-A-C-G-A “
