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Ksivusya [100]
3 years ago
11

An enhancer may increase the frequency of transcription initiation for its associated gene when… (indicate true or false for eac

h statement and explain your answer…) A. …it is located 1000 nucleotides upstream of the gene’s core promoter. B. ...it is located 1000 nucleotides downstream of the gene’s core promoter. C. …it is in the gene’s coding region.
Biology
1 answer:
Olenka [21]3 years ago
4 0

Answer:

it is located 1000 nucleotides upstream of the gene’s core promoter - true

it is located 1000 nucleotides downstream of the gene’s core promoter- true

it is in the gene’s coding region - False

Explanation:

These enhancers are located 50 or more kilobases from the promoter they controlled upstream from a promoter, downstream from a promoter within an intron, or even downstream from the <u>final exon</u> of a gene which can be thousands of bp away from the gene's core promoter and can also occur thousands of nucleotides away from the gene's core promoter needing the activity of a DNA -bending protein that binds to the enhancer changing the shape of the DNA and allow interactions between the activators and transcription factors.

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Initially, little was known about how AIDS was transmitted, and even less was known about the virus that caused it. In 1985, the virus itself was isolated. Following this discovery, Margaret Heckler, the US Human Services Secretary at that time, famously declared, "We hope to have a vaccine [against AIDS] ready for testing in about two years."

Vaccines have worked well against once widespread diseases like smallpox and polio. After the AIDS virus was found, many people, including many scientists, thought AIDS would be added to the list. Vaccines mimic natural infections, during which the body produces antibodies that kill the virus. But unlike smallpox or polio, HIV doesn’t stimulate this kind of response – our immune systems are generally blind to the virus and unable to launch an effective antibody attack. Other challenges that scientists face as they try to create a vaccine include a lack of good animal models to study and the virus's ability to constantly change and mutate. Additionally, although controllers can keep levels of the virus low, no one has ever fully recovered from HIV infection. This means there's no natural, winning strategy for scientists to study and try to elicit.

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In addition to approaches that try to stimulate antibody immunity, researchers are also looking for ways to stimulate cellular immunity, or activate the other weapons in the immune system’s arsenal, like macrophages, natural killer cells, T cells, and more. Alerting the body’s immune system to HIV’s invasion may not prevent infection, but it could inhibit the disease’s progression and keep viral populations so low that there might be less risk of transmission.

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