Answer:
All of the options are true for a MRSA infection.
Explanation:
<em>Staphylococcus aureus</em> is one of the most frequent pathogens causing hospital and community infections. <em>S. aureus</em> can become very easy methicillin resistant (called MRSA isolates) and others beta-lactam antibiotics (are the ones widely used to treat infections) and usually can be resistant to other class of antibiotics, become a very strong bacteria making treatment options very limited. MRSA isolates can rapidly transfer the methicillin resistance to other species of S<em>taphylococcus</em> and some other bacteria. Also <em>S. aureus</em> can acquire other antibiotic resistant genes making a deadly bacterium for its strong resistance. It is in search how the bacterium acquire this antibiotics resistance ( and other virulence factors genes) and the mechanism involve to develop new drugs to treat MRSA infections with the hope that can´t develop resistance to this new drugs.
Oxygen, it takes in Carbon Dioxide and releases Oxygen as a waste :)
Answer:
first one is a point mutation
second is a frame shift by addition
Explanation:
First one : only one letter changes
second one : a letter was added shifting other bases.
Answer:
A. PfEMP1
Explanation:
PfEMP1 stands for <em>Plasmodium falciparum</em> erythrocyte membrane protein-1. These antigens play a very important role in host immune invasion. Production of antibody against PfEMP1 antigens has been shown to contribute to natural immunity.
Malaria is associated with the parasites exhibiting an antigenically distinct <em>Plasmodium falciparum</em> erythrocyte membrane protein-1 subset thereby mediating binding to endothelial receptors.
