The resulting mice will be induced with the changed gene either on or off. The offspring will have altered genes if both the alleles have the trait. This is a heritable change.
Explanation:
Making gene on or off is a process of gene regulation when one of the gene is methylated or histone modification to prevent access to transcription factors hence no expression of the protein. This process is called epigenetics.
The resulting progeny in mice will receive the allele of on or off the gene. Such genes with altered allele are also called as 
The altered genes will be expressed in the resulting progeny of mice.
The epigenetic genes can be reverted with environmental effects in the offspring paramutable alleles. They are heritable.
 
        
             
        
        
        
They are ineffective against viral infections because viruses can not be treated with antibiotics.
        
             
        
        
        
Charles Darwin noticed that among the same species there were variations in traits based upon geographical location. These changes may have evolved from climate differences, predator/prey relationships and food sources. This particular experiment was intended to duplicate natural selection by demonstrating the concepts with artificial selection. The result was that over time, new traits could be bred for. Although there may be more than one correct answer here, the best answer is the first; desirable traits can be selected and bred in offspring.
        
             
        
        
        
Answer:
Here the fucose is linked to galactose by alpha1,2 glycosidic linkage.
Explanation:
Fucose is a deoxy sugar consist of 6 carbon atoms which means fucose is a hexose sugar.
    Fucose does not contain -OH group at C6 carbon that"s why it is termed as deoxy sugar.
      Fucose can link to both Nactylglucosame by alpha-1,6 glycosidic linkage and to galactose by alpha-1, glycosidic linkage  generates the H antigen.
    As donor is blood type B that means fucose is linked to galactoseof H antigen by alpha 1,2glycisidic linkage.