Phenotype is referred to as the expressed trait that is caused by a certain combination of alleles.
<h3>What is phenotype?</h3>
Phenotype is referred to as the expressed trait that is caused by a certain combination of alleles. We will recall that the HbS allele is responsioble for the anemia that results from sickle shaped red blood cells.
This phenotype is both harmfull and benefical in the sense that it leads to the agglutination of the red blood cells in individuals that have this phenotype and eventual crisis. Also, it is beneficaila in that nthose who possess this phenotype do not suffer from malaria in the tropics.
Learn more about HbS allele: brainly.com/question/12347919
Answer:
<h3>Yes</h3>
She always prepared food.
Answer:
1. The difference between the normal hemoglobin protein DNA sequence and the sickle cell hemoglobin DNA sequence is a base to base shift, in this case adenine (GAG) to thymine (GTG).
2. The difference affects the amino acid sequence of the protein by replacing glutamic acid (Glu) with valine (Val).
Explanation:
In sickle cell anemia, a change in the DNA nucleotide sequence is observed, where adenine is substituted by thymine, whose expression is the change in the amino acid sequence of globine β, incorporating valine instead of glutamic acid. This represents a molecular mutation - point mutation - by subtitution, which corresponds to missense mutation.
<u>Normal hemoglobin protein in a RBC</u>
DNA CTG ACT CCT GAG GAG AAG TCT
Amino acids Leu Thr Pro Glu Glu Lys Ser
<u>Sickle cell hemoglobin protein in a RBC</u>
DNA CTG ACT CCT <em>GTG</em> GAG AAG TCT
Amino acids Leu Thr Pro <em>Val</em> Glu Lys Ser
When GAG is transcribed to mRNA, the CUC codon is obtained, which codes for glutamic acid. Thymine substitution causes the DNA sequence to change to GTG, which is transcribed as CAC, the codon that encodes the amino acid valine. The <u>change from glutamic acid to valine in β-globin causes an altered hemoglobin, giving the abnormal erythrocytes observed in sickle cell disease</u>.
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