Answer:
Can you restate your question. That is way to broad
Explanation:
Answer:
characterized by presence or absence of antigens
the blood types are A, B, O, AB
Explanation:
There are two antigens and two antibodies that are mostly responsible for the ABO types. The specific combination of these four components determines an individual's type in most cases. Erythrocytes and serum were related to the presence of antigens on these erythrocytes and antibodies in the serum. these antigens are A and B, and depending upon which antigen the erythrocytes express, blood either belonged to blood group A or blood group B. A third blood group contained erythrocytes that reacted as if they lacked the properties of A and B, and this group was later called "O" blood group. The fourth blood group AB, was added to the ABO blood group system. These erythrocytes expressed both A and B antigens.
Blood group Antigen present on RBC Antibodies in serum Genotype(s)
A antigen A anti-B AA or AO
B antigen B anti-A BB or BO
AB both A and B antigen none AB
O none anti-A and anti-B OO
Answer:
Only some species may survive the event
Explanation:
Evolution is most likely to occur when catastrophic events alter community composition. Catastrophic global event can influence that only the hardiest, generalist species may survive. Specialists may be left without food and starve.
Answer:
The possible fate of the cell that it may turn cancerous.
Explanation:
The cells present in the body generally work in harmony. However, if a cell attains a mutation, it can make it proliferate in the case when it should not do, and make it thrive in the case when other cells are dying. Due to proliferation, the unusual cell produces more abnormal cells also known as cancerous cells. These cancerous cells become more favorable in comparison to the normal cells due to the phenomenon of natural selection. These cells eventually result in a lethal form of tumors.
In the normal cells, the destructed gene or the damaged cells get repaired easily, in case if the damage is worse the cell dies. A protein known as p53 helps in repairing damaged cells or kills them if the damage is too severe. But in the case of cancer cells, the p53 protein does not work appropriately as they possess a mutated or changed form of p53 protein. Thus, in the case of cancerous cells, the rate of repair lags behind the rate of mutation, which makes the cancer cells thrive and increase in numbers resulting in further destruction.
