I believe that a good way to summarize all the processes is metabolism.
Metabolism is the sum of all the (chemical) processes starting at the beginning of an organisms' life and ending at death.
I think C, the egg is larger so contains more mitochondria. Reason is we maily obtain the mtDNA from our mothers :v
You did not post the scenario
Answer:
1. It is now illegal to import or purchase Burmese pythons in Florida. Probably, at some point, python owners who no longer wanted to care for them let them go in the Everglades. By the mid-1990s, the pythons had established a breeding population.
2. There have been no human deaths from wild-living Burmese pythons in Florida. Overall, the risk of attack is very low. ... The simplest and most sure-fire way to reduce the risk of human fatalities is to avoid interacting with a large constrictor.
Explanation:
Burmese pythons are not poisonous snakes, however they are constrictors, coiling around their prey and squeezing the life out of it. The officials in the state of Florida are extremely concerned about the invasion of these large snakes and their ability to take over most of the Everglades.
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
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