Patella is the word you are looking for
1. Action potential reaches the axon terminal and depolarizes it.
2. Depolarization opens voltage-gated calcium channels, enabling influx of Ca into the neuron.
3. Calcium binds to specialized proteins on vesicles (containing pre-made acetylcholine) and triggers them to fuse with the neuron membrane at the synapse.
4. Exocytosis of acetylcholine into the synaptic cleft occurs.
5. Acetylcholine diffuses across the synapse and binds to nicotinic receptors on the end plate of the myocyte.
6. Activated nicotinic receptors, themselves ion channels, cause cation influx into the myocyte and generate an end plate potential. This eventually gives rise to the full depolarization within the myocyte that enables contraction.
Answer;
Inflammatory
The physiologic process that furthers a burn injury is inflammatory process.
Explanation;
Initial inflammatory response to a burn injury results from activation of the signalling pathways in immune cells by intracellular molecules that are released from the damaged cells.
Answer:
from the axon hillock toward the axon terminals.
Explanation:
Axon hillock is a cone-shaped elevation of an axon. It is the region where axons join the cell body. In most neurons, nerve impulses arise at the junction of the axon hillock and the initial segment of axons. This region is called the trigger zone. From this region, a nerve impulse travels towards axon terminals. Axon terminals may have synaptic end bulbs at their tips. As nerve impulse travel from axon hillock to the axon terminal, synaptic vesicles having neurotransmitters are released into the synaptic cleft.
