1. Explain why neither cyclins nor kinases alone can cause a cell to progress through the cell cycle.
As cyclin accumulates, it activates their kinases that turn on the pathway to mitotic spindle formation, and so on.
2. How do controls of the cell cycle protect multi-cellular organisms from accumulating large numbers of damaged or defective cells?
The checkpoint control is responsible for multi-cellular organisms for not accumulating large numbers of damaged or defective cells. Checkpoint controls consist of proteins that detect mistakes and damage and quickly halt the cell cycle until repairs are made. When this occurs, the cell is said to be in cell-cycle <span>arrest.
</span>3. What is the difference between a cancerous tumor and metastasis?
Cancer is cause by mutations in the genes that encode these proteins can lead to uncontrolled growth. Cancer is when there is uncontrolled cell growth and reproduction. Metastasis is caused by tumors when they grow and interfere with the surrounding tissue or cells and break off and spread around the body. Cancerous tumors cause metastasis, and tumors are caused by mutations in genes that lead to uncontrolled growth.
4. What are the functions of tumor-suppressor genes and protoncogenes in noncancerous cells?
The genes that encode the checkpoint proteins are called tumor suppressors because they suppress the development of cells into tumors. If mutations inactivate these genes, the cell-cycle break is removed with or without a signal from the outside. Proto-oncogene’s are involved in promoting cell division, mutations can cause them to become oncogenes, or cancer genes which stimulate cells to leave G0 and divide whether or not it is a signal.
Carbon is an integral part of many biological processes. Living things use carbon throughout their system. Plants need carbon to go through photosynthesis. People give off carbon through cellular respiration. Hope this was somewhat helpful
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Answer:
C) Through genomic imprinting, methylation regulates expression of the paternal copy of the gene in the brain.
Explanation:
The pattern of gene expression wherein either paternal or maternal gene is expressed in specific cells while the other one is prevented from expression is known as genomic imprinting.
In the given example, the maternal copy of the gene on chromosome 15 is expressed in brain cells while its paternal copy is not expressed in these cells. Hence, the pattern of expression of this gene is regulated through genome imprinting. One of the mechanism is methylation of cytidine residues of CpG islands of the DNA that are more frequently present within promoters of the genes.
When the cytidine residues of these sequences are methylated into 5-methylcytidine, the transcription factors do not bind to these promoters preventing the expression of these genes.
Hence, methylation of cytidine residue in CpG islands of the promoters of the gene present on chromosome 15 could have silenced its expression in brain cells.
