During the krebs cycle NADH (Nicotinamide adenine dinucleotide) and FADH (<span>Flavin adenine dinucleotide) are made and used in the electron transport chan. Hope this helps!</span>
D i believe, i could be wrong though
<span>All the options (heat, pressure, deposition and chemical environment) are correct.<span>
Metamorphic rocks are formed when pre-existing rocks are subjected to heat, pressure and hot mineral rich fluids which cause physical, chemical and mineralogical changes to the rock. Metamorphic rocks are grouped by chemical, texture and mineralogy. The common metamorphic rocks include schist, marble, and gneiss.
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Non-random mating is assortative mating. It is a pattern and form of sexual selection in which individuals with similar phenotypes mate with one another more frequently than would be expected under a random mating pattern. non-random mating can act as an ancillary process for natural selection to cause evolution to occur. It’s also bad for evolution because any departure from random mating upsets the equilibrium distribution of genotypes in a population. Recombination is a process by which pieces of DNA are broken and recombined to produce new combinations of alleles. recombination is important to somatic cells in eukaryotes because it can be used to help repair broken DNA. recombination by itself does not cause evolution to occur. Rather, it is a contributing mechanism that works with natural selection by creating combinations of genes that nature selects for or against. Non-random mating affects the evolution more than recombination
When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
