Protein synthesis in the ribosomes
Answer:
38
Explanation:
In eukaryotic cells, the maximum production of ATP molecules generated per glucose molecule during cellular respiration is 38, i.e., 2 ATP molecules from glycolysis, 2 ATP molecules from the Krebs cycle, and 34 ATP molecules from the Electron Transport Chain (ETC). <em>In vivo</em> (i.e., in the cell), this number is not reached because there is an energy cost associated with the movement of pyruvate (CH3COCOO−) and adenosine diphosphate (ADP) into the mitochondrial matrix, thereby the predicted yield is approximately 30 ATP molecules per glucose molecule. In aerobic bacteria, aerobic respiration of glucose occurs in the cytoplasm (since bacteria do not contain membrane-bound organelles such as mitochondria), and thereby, in this case, it is expected that aerobic respiration using glucose yields 38 ATP per glucose molecule.
Answer:
All of the options are true for a MRSA infection.
Explanation:
<em>Staphylococcus aureus</em> is one of the most frequent pathogens causing hospital and community infections. <em>S. aureus</em> can become very easy methicillin resistant (called MRSA isolates) and others beta-lactam antibiotics (are the ones widely used to treat infections) and usually can be resistant to other class of antibiotics, become a very strong bacteria making treatment options very limited. MRSA isolates can rapidly transfer the methicillin resistance to other species of S<em>taphylococcus</em> and some other bacteria. Also <em>S. aureus</em> can acquire other antibiotic resistant genes making a deadly bacterium for its strong resistance. It is in search how the bacterium acquire this antibiotics resistance ( and other virulence factors genes) and the mechanism involve to develop new drugs to treat MRSA infections with the hope that can´t develop resistance to this new drugs.
"REPRODUCTIVE ISOLATION" needs to occur for speciation to happen.....
*High ventilation/low perfusion that Increased the cardiac output Decreased of the pulmonary vascular and the resistance and *Pulmonary hypertension,*Reduced the left ventricular preload
Pulmonary hypertension is happens when the pressure in the blood vessels and leading from the heart and the lungs is too high. With the pulmonary hypertension, the blood vessels in the lungs to develop the increased amount of muscle in wall of the blood vessels
It's a serious condition which can be damage the right side of the heart. and The walls of the pulmonary and the arteries to become the thick and the stiff, and that cannot be expand as well they allow blood through.
To know more about Pulmonary hypertension visit:
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