Answer:
All of the above
Explanation:
ATP synthase is a transmembrane protein enzyme. It harnesses the potential energy –proton motive force- created by the development of a proton gradient across a membrane (could be across the intermembrane space in chloroplast and mitochondria). As the H+ ions 'drain' back and pass through their channels in the protein enzyme, the synthase is able to phosphorylate ADP and Pi to form ATP.
These ATPs (from photophosporylation) in light-dependent phase, are used in the catabolism of glucose, in the light-indepedent phase.
This could be that the patients ovaries are unable to produce eggs anymore or have a diminished ovarian reserve and produce few eggs...
Answer:
A. using transitional forms
Explanation:
On Edge 2020
The origin of congenital adrenal hyperplasia occurs when circulating cortisol levels are low.
Congenital adrenal hyperplasia (CAH) is an autosomal recessive disease which results from mutations of genes for enzymes involved in the biochemical steps of production of mineralocorticoids, glucocorticoids or sex steroids from cholesterol by the adrenal glands (steroidogenesis). <span>Low cortisol production results in rising levels of ACTH (adrenocorticotropic hormone) because cortisol usually inhibits ACTH production. This increased ACTH production induces overgrowth (hyperplasia) and overactivity of the steroid-producing cells of the adrenal cortex.</span>
Foxes Hawks and Owls will have to rely on other sources for food and be the most directly effected. You could make the argument that all the organisms will effected as some will see increased predation, while the plants will see a reduction in herbivores eating them.
