B. Lowering taxes in order to stimulate spending. When the economy experiences a downturn, the government is more likely to cut taxes to allow price competitiveness of goods, thereby triggering demand, hence consumption among households. A boost in consumption should translate into an increase in supply that wwould in turn bring about job creation. The negative trend would therefore be reversed.
A major problem to understand and correct the problems with the San Joaquin Valley lack of tools and scientific data on valley fever.
- A. lack of scientific tools and data
<h3>What is valley fever?</h3>
The small town of Avenal, in the San Joaquin Valley, is most affected by an incurable disease known as 'valley fever'. Cases of an incurable disease called "valley fever", are multiplying at an alarming and surprising rate in the southwestern United States.
With this information, we can conclude that San Joaquin Valley, is best known for an incurable disease known as 'valley fever', where there is no scientific data that can explain.
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Answer:
The exoskeleton helps support movement by working with the muscles.
Explanation:
Answer:
Human genome has various replicons.
Explanation:
A replicon which is a unit of DNA, has potential to get replicated independently. So, the DNA replication occurs simultaneously at various replicons as a result of which human genome takes much less time than estimated to get replicated .
In contrast to this, prokaryotes like E.coli have only one origin of replication and thus one replicon due to which replication is restricted to once per cell cycle.
The correct answer is the last statement.
If the regulatory serine is mutated to alanine, then acetyl-CoA carboxylase will get activated spontaneously and will produce malonyl-CoA. The increased concentrations of malonyl-CoA will obstruct the oxidation of fatty acids by preventing the entry of fatty acids into the mitochondria.
It is because the AMP-activated protein kinase phosphorylates the serine residues of acetyl-CoA carboxylase to inactivate it. If a mutation occurs in such residues, then the AMPL cannot phosphorylate acetyl-CoA carboxylase and this enzyme will get activated spontaneously.
In such a situation, there will be more than sufficient production of malonyl-CoA, which will inhibit the admittance of more fatty acid getting inside the mitochondria; this will indirectly prevent the oxidation of fatty acids.
