Answer:
- Interruption in the genetic flow between separated groups
- The emergence of new mutations in each of the groups, and their accumulation in time. Slow and gradual differentiation between populations.
- Genetic divergence by natural selection and reproductive isolation
- Prezigotic isolation mechanisms
Explanation:
Allopatric speciation consists of the geographic separation of a continuous genetic background that can give place to two or more new geographically isolated populations. These separations might be due to migration, extinction of geographically intermediate populations, or geological events. In this speciation, some barriers impede genetic interchange, or genetic flux, as the two new groups that are separated can not get together and mate anymore. These barriers might be geographical or ecological.
The process of allopatric speciation involves different steps that affect organisms:
- The emergence of the barrier.
- Interruption in the genetic interchange
- The occurrence of new mutations and their accumulation in time in each population. Slow and gradual differentiation.
- Genetic divergence by natural selection and reproductive isolation makes it impossible for the two groups to mate even if the barrier disappears.
- Prezigotic isolation mechanisms will be favored by selection if occurs a secondary contact between the new species in formation.
Answer:
The main step to prepare herbarium sheet are:
Collection and pressing of specimens. Fresh materials are pressed in plant press in herbarium press. ...
Drying of specimens. ...
Mounting of specimens on herbarium sheets. ...
Labeling of specimens. ...
Chemical elements are important to living organisms because they make up the organic molecules that are found in all living things
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
The component is called 'resting metabolic rate. It operates when the body is at complete rest. It can also be called basal metabolism.
