Answer:
the chlorophyll in the leaves
Answer:
That the purple trait is dominant and the purple pea plant has a gene type of PP and the white flower has a gene type of pp
ANSWER: The completion of the Human Genome Project
EXPLANATION:
Human Genome Project (HGP) was completed in April, 2003. Genome varies from one individual to another.
The project involved mapping and sequencing of some people and in other to get each chromosome full sequence in individuals.
However, at the beginning of this project, concerns like ownership and privacy of personal genetic information began to spring up. People are afraid that employers may have access to their genetic information and would reject persons with health issues indicated by their unique genes and health insurance companies may also not provide insurance to people that have deficiency.
In the view of this concern, the United States in 1996 passed the Health Insurance Portability and Accountability Act (HIPAA) which guides against the non-consensual and unauthorized release of health information of individuals.
<span>Lafora disease is the most severe teenage-onset progressive epilepsy, a unique form of glycogenosis with perikaryal accumulation of an abnormal form of glycogen, and a neurodegenerative disorder exhibiting an unusual generalized organellar disintegration. The disease is caused by mutations of the EPM2A gene, which encodes two isoforms of the laforin protein tyrosine phosphatase, having alternate carboxyl termini, one localized in the cytoplasm (endoplasmic reticulum) and the other in the nucleus. To date, all documented disease mutations, including the knockout mouse model deletion, have been in the segment of the protein common to both isoforms. It is therefore not known whether dysfunction of the cytoplasmic, nuclear, or both isoforms leads to the disease. In the present work, we identify six novel mutations, one of which, c.950insT (Q319fs), is the first mutation specific to the cytoplasmic laforin isoform, implicating this isoform in disease pathogenesis. To confirm this mutation's deleterious effect on laforin, we studied the resultant protein's subcellular localization and function and show a drastic reduction in its phosphatase activity, despite maintenance of its location at the endoplasmic reticulum.
I got my information from </span>https://www.ncbi.nlm.nih.gov/pubmed/14722920
