Assuming this is supposed to be a multiple choice question- I think the correct answer would be “early onset of spring change in marine ecosystems” because it looks like the question is asking what it wouldn’t affect, and I’m sure if the people had to leave, that animals would’ve all had to leave as well!
C.
The force the wind exerts on the surface of the rock while it blows past it can cause it to erode into irregular shapes.
In spite of the fact that this situation is probably not going to occur, rotenone is poisonous in light of the fact that it meddles with the electron transport chain (ETC) in mitochondria, so NADH would develop as recommended.
Answer:
Density is an intensive property. This means that regardless of the object's shape, size, or quantity, the density of that substance will always be the same. ... It is because density in an intensive property of matter. So they are not considered properties.
Explanation:
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
