Answer:
when the pores are completely filled in the rocks and soil
Explanation:
The zone of saturation is part of the divisions of ground water systems. It is the zone in which the pore spaces within the rock are completely filled with water.
The zone of saturation is where the aquifer lies in a body of water and it is the ground water prospect. Pore spaces can also be filled with hydrocarbons.
Above the zone of saturation is the water table. The water table defines the upper layer of the zone of saturation above which the pore spaces are filled with gas or no water can be found.
The answer is B. Genetic drift greatly affects small populations, but large populations can recover.
The limitations of cell size are:
1) Surface area to volume ratio
2) Nucleo cytoplasmic ratio
3) Fragility of cell membrane
What are the cause of limitations on cell size?
The need for a surface area of sufficient area to allow the cells function→.
→If the surface area of cell increase volume increase to the cube of the linear increase.
→ Nuclie contains information needed for protein synthesis . Some cells reduce this limitation to increase the no. Of nucleus called multinucleated.
Examples are muscle cells.
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Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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Answer:
where the is the picture?
