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2 years ago

How has plant and animal breeding affected humankind and genetics?

1 answer:

5 0

On the downside, certain exaggerated animal traits produced through selective breeding may make it difficult for the animal to live a normal life, and plants propagated through selective breeding may be prone to disease.

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Crickets, grasshopper, cockroaches, and dragonflies are examples of organisms that go through incomplete metamorphosis. A physic

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1 year ago

Sid created a chart to summarize characteristics of satellite motion.

kap26 [50]

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Centripetal force is perpendicular to the velocity of the satellite.

Explanation:

because there is no friction or wind resistance, the speed of the satellite remains the same

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3 years ago

Read 2 more answers

Fill in the blanks.

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1. Parasites

2. herbivores

3. phagocytosis

4. Plant

5. oxygen (O2)

6. gills

7. open

8. urea

9. Single-loop circulatory systems have one direction of blood flow. Typically in fish, oxygen is absorbed in the gills, flows in blood to the body organs, and back to the heart, which pumps it directly back to the gills. In double-loop circulatory systems, the heart has multiples of each chamber. Part of the heart pumps blood to the lungs, to get oxygen. This blood flows back to a different chamber where it then gets pumped to the body. Once its oxygen has been used up, it goes back to the heart to be pumped all over again to the lungs.

10. Freshwater animals have lower water and higher salt concentrations than the environment in their cells. This means water constantly tries to diffuse into the animals, which can bloat them. They have special cell processes that remove extra water.

pennfoster

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2 years ago

Why might scientists use computer models in research instead of conducting an experiment on the real system they would like to s

Usimov [2.4K]

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Using the real system might mess up and using a computer model is clearer and gives a more advanced view

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2 years ago

Each of the four pedigrees that follow represents a human family within which a genetic disease is segregating. Affected individ

Ne4ueva [31]

Answer:

The following four traits are -:

Pedigree 1 - A recessive trait (autosomal recessive) is expressed by pedigree 1.
Pedigree 2- Recessive inheritance is defined by Pedigree 2.
Pedigree 3 - The inheritance of the dominant trait (autosomal dominant) is illustrated by Pedigree 3.
Pedigree 4- An X-like dominant trait is expressed by Pedigree 4.

Explanation:

Explaination of each pedigree chart-

Pedigree 1 demonstrates the recessive trait since their children have been affected by two unaffected individuals. If the characteristics were X-linked, in order to have an affected daughter, I-1 would have to be affected. $X^A$ In this, both parents are autosomal recessive trait carriers, so the child will be affected by a 1/4 (aa)
Recessive inheritance is defined by Pedigree 2. This is X-related inheritance as autosomal recessive inheritance has already been accounted for in part 1. This inference is confirmed by evidence showing that the father (I-1) is unaffected and that only the sons exhibit the characteristic in generation II, suggesting that the mother must be the carrier. The individual I-2 is a carrier for this X-linked trait. A typical Xa chromosome is attached to the unaffected father (I-1), so the chance of carrier II-5 is 1/2. Probability of an affected son = 1/2 (probability II-5 is a carrier) x 1/2 (probability II -5 contributes ( $X^A$ ) x 1/2 (probability of Y from father II-6) = 1/8. An affected daughter's likelihood is 0 because a typical $X^A$ must be contributed by II-6.
The inheritance of the dominant trait is demonstrated by Pedigree 3 because affected children still have affected parents (remember that all four diseases are rare). The trait must be autosomal dominant because it is passed down to the son by the affected father. There is a 1/2 risk that the heterozygous mother (II-5) would pass on mutant alleles to a child of either sex for an autosomal dominant feature.
Pedigree 4 is an X-linked dominant function characterized by the transmission to all of his daughters from the affected father but none of his son. On the mutant X chromosome, the father (I-1) passes on to all his daughters and none of his sons. As seen by his normal phenotype, II-6 therefore does not bear the mutation. An affected child's likelihood is 0.

In the question the pedigree chart was missing ,hence it is given below.

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2 years ago