The answer is: It spins a sample to separate based on density.
When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
Answer:
a. It's a carrier protein doing facillitated diffusion
Explanation:
Facilitated diffusion may be defined as the transportation of molecules from a high concentration to a low concentration that is down the concentration gradient without input of the energy. Whereas the active transport is transportation of the molecules from the low concentration to the high concentration which is against concentration gradient by the membrane protein.
The glucose transportation process requires membrane protein for the transport and does not require any energy. Therefore, it is carrier protein which is doing the facilitated diffusion.
Mutualism since the clown fish and the anemone both benefit from each other.
Scientists have concluded that the average human body contains approx. 37.2 trillion cells. However it depends upon your size compared an average human being. :)<span />