The main mechanism that can cause loss of fluid in the body would be from sweating and from urinating. Pathological causes that might cause loss of body fluid like diarrhea and bleeding.
Drugs that induce sweating is called diaphoresis. Drugs that induce urine production called diuretic.
If the question is ask for the name of condition where the body loses fluid it can be dehydrated or shock(more severe than dehydrated).
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There are three basic foot shapes, each of which is defined by a different configuration of the toes. When choosing pointe shoes, the first step is to look at your toes, which can be one of three shapes—Egyptian, Grecian or Giselle. Once you know your toe shape, you can narrow down which shoes will fit you best—tapered, somewhat tapered or square.
No, Stomach pepsin digests about 20% of the proteins the rest is digested in the small intestine enzymes but yes protines are digested by hydrolysis
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Skills and Abilities A clinical research coordinator needs to be able to handle multi-tasking and multiple projects. Ensure that you have skills to manage and organize your schedule and be able to discuss how you handle prioritizing tasks under pressure and deadlines. Being detail-oriented is important.
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Multiple sclerosis (MS) is the most common demyelinating and an autoimmune disease of the central nervous system characterized by immune-mediated myelin and axonal damage, and chronic axonal loss attributable to the absence of myelin sheaths. T cell subsets (Th1, Th2, Th17, CD8+, NKT, CD4+CD25+ T regulatory cells) and B cells are involved in this disorder, thus new MS therapies seek damage prevention by resetting multiple components of the immune system. The currently approved therapies are immunoregulatory and reduce the number and rate of lesion formation but are only partially effective. This review summarizes current understanding of the processes at issue: myelination, demyelination and remyelination—with emphasis upon myelin composition/architecture and oligodendrocyte maturation and differentiation. The translational options target oligodendrocyte protection and myelin repair in animal models and assess their relevance in human. Remyelination may be enhanced by signals that promote myelin formation and repair. The crucial question of why remyelination fails is approached is several ways by examining the role in remyelination of available MS medications and avenues being actively pursued to promote remyelination including: (i) cytokine-based immune-intervention (targeting calpain inhibition), (ii) antigen-based immunomodulation (targeting glycolipid-reactive iNKT cells and sphingoid mediated inflammation) and (iii) recombinant monoclonal antibodies-induced remyelination.Keywords: calpain, central nervous system, demyelination, fingolimod, glycolipids, lipids, multiple sclerosis, myelin, myelination, NKT cells, oligodendrocytes, remyelination, T cells
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