Answer:
See the answer below.
Explanation:
Antibiotic-producing bacteria are generally known to have a mechanism that enables them to be resistant to their own antibiotics. The mechanism that enables them to be resistant to their own antibiotic depends largely on the mode of action of the antibiotic substance.
Some of the popular mechanisms used by bacteria to counter their own antibiotic substance include a mutation in the target gene, production of enzymes that inactivate the antibiotic compounds, or efflux of the compounds.
<u>In the case of </u><u><em>Streptomyces griseus</em></u><u>, the inactivity of streptomycin has been linked with the production of a phosphatase inhibitor that prevents streptomycin from getting access to the target site. Hence, the organism is not harmed by its own antibiotic.</u>
Answer:
G1 phase cell cycle arrest and apoptosis
Explanation:
According to Yan and colleagues 2009 article, cells that were transfected with microRNA-1/206 showed cell cycle arrest at the G1 phase and showed an increase in apoptosis (programmed cell death) which is important for synthesis of mRNA and protein. These processes have a direct effect on cell proliferation by decreasing it.
Answer: Without oceans, there would be more evaporation and more precipitation on Earth
