Answer: If a mutation occurred in Silencer of Death Domain (SODD) that prohibited its interaction with the DD of TNF receptor, the TNF receptor would evoke its response by binding to a transmembrane receptor, TNFR1.
Explanation: Binding to TNFR1 inhibits the recruitment of cytoplasmic signaling proteins to TNFR1 to prevent recruitment of a number of protein (TRADD) or prevent spontaneous aggregation of the cytoplasmic death domains of TNFR1 molecules.
SODD mechanism is useful in preventing unwarranted TNFR1 activation in the process of apoptosis.
Answer:
The correct answer will be-
1. decrease in blood volume
2. secretion of renin from juxtaglomerular apparatus
3. production of angiotensin II
4. secretion of aldosterone
5. sodium reabsorbed from distal tubules and collecting ducts
Explanation:
The RAAS pathway or Renin-angiotensin II-aldosterone pathway gets activated in the human body to maintain the osmolarity and blood volume.
When the receptors in the blood sense the low volume of water in the body sends signals to the brain which activates the secretion of the renin from the juxtaglomerular cells. Renin helps converts the inactive angiotensin to angiotensin I and II which help reabsorption of sodium ions. This activates the production of aldosterone which increases the reabsorption of the sodium ions into distal and collecting ducts.
Trans fats.
Unsaturated fat is GOOD fat, saturated fat is bad fat as well as trans fats.
Answer:
Oxygen and pyruvate.
Explanation:
The krebs cycle is a stage of cellular respiration and occurs in aerobic organisms. In this case, we can say that the Krebs Cycle only occurs in the presence of oxygen, being essential for the completion of cell respiration.
The Krebs cycle only occurs after the completion of glycolysis, as it needs to be initiated by pyruvate, which is a molecule resulting from glycolysis. In this case, in addition to oxygen, the presence of pyruvate is essential for the krebs cycle to occur.
