Stimulant speeds up body functions but depresses brain activity
Inhalant dissolves the fatty tissue in the brain
Steroids influences moods in the limbus system
Evidence suggests that cAMP response-element-binding protein (CREB) may be a final common target of antidepressant treatment.
Antidepressant medications work by binding to a number of monoamine neurotransmitter receptors and activating the cAMP signal transduction pathway. Chronic antidepressant therapy has been linked to the cAMP signalling system. Increases in cAMP cause the cAMP response element-binding protein(CREB) to be phosphorylated more frequently via increased receptor adenylate cyclase activity .
CREB mediates transcriptional responses to increased cAMP levels by attaching to cAMP response element (CRE) sites.In order to prevent recurring episodes, treatment for depression should maintain therapeutic efficacy while also addressing behavioural symptoms and indicators of the illness. One important modulator of the transcriptional effects of antidepressants may be CREB.
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Answer:
Oxytocin, a posterior pituitary hormone. (Ans A)
Explanation:
Oxytocin is known as a posterior pituitary hormone that helps to stimulate strong contractions during labor pain and help to dilate the cervix, push the baby downward and out through the birth canal, drag out of the placenta, and restrict the bleeding at the placental site.
During the birth process, against the cervix, the pressure of the baby and then against the pelvic floor tissues helps to promote contractions and oxytocin hormone. Low levels of oxytocin hormone can cause several problems such as after birth excessive bleeding at the placenta site, contractions slow or stop and making labor time take longer, etc.
