The probability is 1/16 that a gamete will receive only paternal chromosomes. In life
cycles that alternate between haploid and diploid stages, fertilization doubles
the number of chromosomes per cell, The pairing of chromosomes along
their lengths , which is essential for crossing over is referred to as synapsis
The monomer of a nucleic acid (DNA is made up of multiple nucleic acids) is a nucleotoid.
Light is the energy source in Photosynthesis!
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Answer:
No, telomerase is not an oncogene. It prevents the senescence that would occur due to shortened telomeres, but the cell proliferation might still be mitogen-dependent.
Explanation
telomerase is not responsible for causing deregulation while oncogenes cause deregulation .
Telomeres length shorten after the cell division which stops them to divide again and cell die.
Telomerase prevents this decline in some kinds of cells, including stem cells, by lengthening telomeres, and the hope was that activating the enzyme could prevent senescence.