Answer:
A. If the aerobic pathway—cellular respiration—cannot meet the energy demand, then the anaerobic pathway—lactic acid fermentation—starts up, resulting in lactic acid buildup and "oxygen debt."
D. The rate of energy demand determines how the muscles will obtain energy, either from cellular respiration or from lactic acid fermentation if not enough oxygen is present.
Explanation:
It is important to consider that Kenny hikes all day but at a steady pace, whereas Janelle runs very fast. So Kenny's case, the supply of oxygen is sufficient to maintain aerobic respiration within the muscle cells. During this process only CO2, Water, and ATP are produced; therefore, there is no oxygen debt. We should keep in mind that the body shifts to anaerobic metabolism only when the supply of oxygen is limited.
In Janelle's case, running fast would need energy at higher rates and the supply of oxygen would not be sufficient to generate a high amount of ATPs. Therefore, to compensate for this deficiency, cells will start fermenting glucose to lactic acid and produce ATP and maintain energy demands. This lactic acid causes fatigue and this is why Janelle has aching and breathing hard. Breathing hard is also automatic reflux to inhale more oxygen and meet oxygen demands but even breathing hard would not be able to make it and the body will shift to anaerobic respiration automatically.
Answer:
the answer is A. E. coli B
Explanation:
The multiplicity of infection (MOI) refers to the ratio between the numbers of viruses used to infect <em>E. coli</em> cells and the numbers of these <em>E. coli </em>cells. Benzer carried out several experiments in order to define the gene in regard to function. Benzer observed that <em>E. coli </em>strains with point mutations could be classified into two (2) complementary classes regarding coinfection using the restrictive strain as the host. With regard to his experiments, Benzer observed that rII1 and rII2 mutants (rapid lysis mutants) are complementary when they produce progeny after coinfect E. coli K (where neither mutant can lyse the host by itself). The rII group of mutants studied by Benzer does not produce plaques on <em>E. coli</em> K strains that carry phage λ (lysogenic for λ), but they produce plaques on <em>E. coli</em> B strains. This study showed that rIIA and rIIB are different genes and/or cistrons in the rII region.
