Answer:
because it would be dangerous to do it by hand or skin.
Explanation:
enzymes are proteins that speeds up the rate of chemical reaction in a living organism . An enzyme acts as catalyst for specific chemical reaction, converting a specific sets of reactants called substrates into specific products with out enzymes life would not be exist
Explanation:
Enzymes are identified to catalyze more than 5,000 biochemical reaction types. Most enzymes are proteins, although a few are catalytic RNA molecules. Enzymes' specificity appears in their unusual three-dimensional structures. Like all enzymes, enzymes improve the reaction rate by decreasing its activation potential.
Answer:
Answer:
Explanation:
1. Monoculture
2. Erosion
3. Desertification
4. Bio-logical Magnification
5. Bio- diversity
6. Ecological hot spot
7. ecological footprint
8. Ozone Layer
9. Aquaculture
10. Global Warming
Explanation:
Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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Answer:
One reason is that there was too much pressure in the bottom of the ocean, from all the water pressing together.
hope this helps :)
Explanation:
