Answer:
BbxBb.
Explanation:
If Bb gene is crossed with Bb, the offspring is produced with normal hearing because both the genes are recessive in which the deafness gene hide and the hearing gene is dominant so the offspring can hear sounds clearly. The dominant A gene is responsible for deafness and we can see the offspring BbxBb in which no dominant gene is present. The other three offspring are deaf because of the presence of dominant A gene in their genetic makeup.
He would say that Lamarck's theory is wrong. Lamarck's theory stated that traits that are used are passed on to the offspring. In other words, if an organism changes during its lifetime in order to adapt to its environment, then its changes will be passed on to its offspring. This is wrong because this means that organisms pass on traits based on genetic information and not based on the environment of the offspring.
Hope this helps.
Producer.
I remember it by thinking: "auto means automatic, so autotrophs automatically make their own food, so they must be producers"
Answer:
Examples of environmental factors that may alter salivary peroxidase include periodontitis, oral hygiene, presence of heavy metal ions, bacteria (e.g., <em>Streptococcus gordonii</em>), anaerobic conditions, temperature, pH, etc.
Explanation:
Peroxidase is an enzyme found in all aerobic cells that act to convert toxic hydrogen peroxide (H2O2) into dioxygen (O2) and water (H2O). This enzyme plays an important non-specific defensive role against proliferating micro-organisms that cause periodontal diseases such as periodontitis, which is a serious inflammatory disease affecting the tissues around the teeth. The most common environmental factors influencing the development of periodontitis include oral hygiene, smoking and age. In this regard, it has recently been shown that there is a positive correlation between salivary peroxidase activity and periodontal health, especially in non-smoker individuals. In consequence, it is expected that smoker individuals are more prone to suffer periodontal diseases by reduction of the salivary peroxidase levels.
