Answer:
A virus attaches itself to cells and usually reprograms them to reproduce itself. Also, unlike bacteria, most viruses do cause disease. Some virus-caused diseases include the common cold, AIDS, herpes, and chickenpox
Explanation:
Answer:Insulin is a horome that your pancreas make to allow to use glucose
Explanation:
Really just cells ..
But not really just cells
Well, Weight and the Planets are measured by kilograms. The SI unit of weight is Newton.
Since weight of a body is nothing but Mass of the body multiplied by acceleration due to gravity acting on it, the SI unit of weight will be the same as the SI unit of force (mass x acceleration), that is, Newton.
However, you may note that since the acceleration due to gravity is different on different heavenly bodies, the value of weight will be different on different bodies, unlike the mass, which is the same throughout the universe.
I hope this helps! :)
Lean persons may acquire a lipid profile with raised LDL- and HDL-cholesterol levels and reduced triglycerides when they follow carbohydrate-restricted diets (CRDs) (TGs). This lipid profile's intensity is correlated with BMI, therefore people with lower BMIs show greater rises in both LDL-C and HDL-C. Lean Mass Hyper-Responders (LMHR) is a subset of people who, while having normal pre-diet LDL-C, as shown by the inverse relationship between BMI and changes in LDL-C and HDL-C on CRD, respond to weight loss by increasing their HDL-C and LDL-C levels. revealed a substantial hyperlipidemic response to a CRD, with mean LDL-C and HDL-C levels rising to 320 and 99 mg/dL, respectively, in the context of mean TG of 47 mg/dL, as opposed to non-LMHR (mean values of 148 and 145 mg/dL, respectively). Again, with very normal pre-diet LDL-C and the absence of genetic evidence suggestive of familial hypercholesterolemia in those who have been examined, some LMHR may have LDL-C levels above 500 mg/dL.With carbohydrate restriction in lean individuals, the increased dependence on fat as a metabolic substrate drives increased hepatic secretion and peripheral uptake of TG contained within very low-density lipoproteins (VLDL), resulting in marked elevations of LDL-C and HDL-C and low TG, according to the Lipid Energy Model (LEM), which aims to explain this metabolic phenomenon. Here, we go through the LEM's main characteristics. We examine various lines of existing research that support the model and make recommendations for how to test its predictions.
To learn more about the lipid Energy Model here:-
brainly.com/question/17412429?referrer=searchResults
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