The correct answer is false. Insulin speeds up glucose transport across cell membranes and promotes glycogen synthesis and slows down glycogen breakdown.
Adipokines, proinflammatory substances, and free fatty acids are released by adipose tissue, an endocrine organ that affects both glucose and lipid metabolism. These substances reduce muscle ATP synthesis and glucose metabolism, encourage the synthesis of harmful lipid metabolites, and change insulin signaling. Insulin affects adipose tissue in two ways: 1) by increasing glucose absorption and triglyceride synthesis, and 2) by reducing triglyceride hydrolysis and the release of FFA and glycerol into the bloodstream. Elevated plasma FFA levels have been demonstrated to impair muscle insulin signaling, promote hepatic gluconeogenesis, and impair glucose-stimulated insulin response. Adipose tissue insulin resistance, which is the impaired suppression of lipolysis in the presence of high insulin levels, has been linked to glucose intolerance.
Learn more about glycogen here:
brainly.com/question/2009860
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Survival??
That’s a complete guess
When the cells undergo late apoptosis, the membrane structure is destroyed and the nuclear structure can be selectively visualized by Hoechst 33342/PI.
https://www.creative-bioarray.com/support/double-staining-apoptosis-assay-hoechst33342-pi.htm
Active transport moves from low to high
