Patty (age 7) has symptoms that include a skin rash, fever slowed growth, fatigue, and swelling in the joints. She was diagnosed as having juvenile rheumatoid arthritis.
What is juvenile rheumatoid arthritis described as?
Juvenile rheumatoid arthritis is the most common kind of arthritis in children. It is characterized by heat and discomfort and causes the joints to expand. The duration of acute arthritis can range from a few weeks or months to years or even a lifetime. It can also be persistent. IA types include autoimmune and autoinflammatory illnesses. This suggests that the immune system, which is meant to fight off viruses and pathogens, becomes confused and attacks the body's cells and tissues. The doctor can suggest blood testing for C-reactive protein and erythrocyte sedimentation rate. These blood tests evaluate inflammatory markers or markers of inflammation.
To learn more about Juvenile rheumatoid arthritis, click below
brainly.com/question/13158067
#SPJ4
Answer:
4 possible combinations of gametes
Explanation:
The animal is considered an heterozygotic dihybrid because it involves two genes that have different alleles for both genes. The dihybrid animal undergoes meiosis to produce gametes. According to Mendel in his laws of segregation and independent assortment, the alleles of the two genes separate into gametes independently of one another to produce 4 types of gametes.
Number of possible combinations of gametes formed by an heterozygote can be determined by using 2^n, where n is the number of characters. Number of characters or genes involved in this case is 2, Hence, 2^2 = 4 gametes.
The allelic combination in the four gametes are: AB, Ab, aB, ab.
Answer:
In the lysogenic cycle, the viral genome gets incorporated into the host genome and replicates with the host genome. In the lysogenic cycle, no proteins and enzymes of the virus are formed while in the lytic cycle protein of viral capsid form and new phages generate in the host cell.
So as the viral genome integrates into the host genome in the lysogenic cycle, therefore, it is difficult for any drug to differentially act on the viral genome. So there are great chances for drugs to target the host genome in the host cell.
But in virus that replicates through lytic cycle drugs can selectively act on viral enzyme and proteins. Therefore it is difficult to make a drug that affects lysogenic virus.
Answer:
Nowadays, if a cell cannot express the AURKA gene, malignant neoplasms or cellular malignancies can be generated.
since it is related to the KRAS gene.
There is some scientific evidence or research that maintains that FOSL1 acts on another gene, AURKA, whose expression to date was thought to be independently regulated by the KRAS oncogene, and for which clinical trials are currently underway with a drug that inhibits it. .
Explanation:
Aurora kinase A also known as serine / threonine-protein kinase 6 is an enzyme that in humans is encoded by the AURKA gene.
They find differnt ways to survive like food shelter hiding from preditors etc
