Wild Bedbugs become insecticide resistant because of the mutations and natural selections.
<h3><u>Explanation</u>:</h3>
As the huge amount of pesticides and insecticides are sprayed in the rooms for cleaning, the pests and insects like bedbugs dies in huge portions because of the toxin. But some of the bedbugs remain alive as they have mutations that help them to detoxify the toxins given, or bypass the metabolic processes so that the toxins don't hamper them much.
Now as the population becomes very small(bottle neck effect), the nature selects these organisms over the other to propagate more sufficiently and enormously. As the nutrients and supplies are also available, so the bedbugs don't suffer any lack of nutrition which can be a determining factor of their population.
Thus the wild bedbugs become resistant to insecticides while the experimental one remain succeptible to insecticides.
Answer:
The difference between Prokaryotic cell and Eukaryotic cell are;
![\begin{array}{lll}&\underline {\text {Prokaryotic}}& \underline {\text {Eukaryotic}}\\ \\ \text {Nuclear \ membrane \ and \ nucleus} & \text {No \ true \ nucleus \ or} & \text {Has \ a \ true \ nucleus} \\& \text {nuclear \ membrane} & \text {nucleoli \ and \ nuclear \ membrane}\\\\ \text {Membrane \ bound \ cell \ organelle} & \text {None &Present} \\\\ \text {Chromosome \ number \ and \ shape} & \text {One} & \text {Several, more \ than \ one} \end{array}](https://tex.z-dn.net/?f=%5Cbegin%7Barray%7D%7Blll%7D%26%5Cunderline%20%7B%5Ctext%20%7BProkaryotic%7D%7D%26%20%5Cunderline%20%7B%5Ctext%20%7BEukaryotic%7D%7D%5C%5C%20%5C%5C%20%5Ctext%20%7BNuclear%20%5C%20membrane%20%5C%20and%20%5C%20nucleus%7D%20%26%20%5Ctext%20%7BNo%20%5C%20true%20%5C%20nucleus%20%5C%20or%7D%20%20%20%20%26%20%5Ctext%20%7BHas%20%5C%20a%20%5C%20true%20%5C%20nucleus%7D%20%5C%5C%26%20%5Ctext%20%7Bnuclear%20%5C%20%20membrane%7D%20%26%20%5Ctext%20%7Bnucleoli%20%5C%20and%20%5C%20nuclear%20%5C%20membrane%7D%5C%5C%5C%5C%20%5Ctext%20%7BMembrane%20%5C%20bound%20%5C%20cell%20%5C%20organelle%7D%20%26%20%5Ctext%20%7BNone%20%26Present%7D%20%5C%5C%5C%5C%20%5Ctext%20%7BChromosome%20%5C%20number%20%5C%20and%20%5C%20shape%7D%20%26%20%5Ctext%20%7BOne%7D%20%26%20%5Ctext%20%7BSeveral%2C%20more%20%5C%20than%20%5C%20one%7D%20%5Cend%7Barray%7D)
Explanation:
Prokaryotic cells
The plasma membranes surround prokaryotic cells, however, within their cytoplasm, there are no organelles bound to the membrane and there is no nucleus in the prokaryotic cell which differentiates it from the eukaryotic cells
Eukaryotic cells
Eukaryotic cells also have plasma membranes, ribosomes and cytoplasm. Eukaryotic cells also have membrane-bound nucleus, membrane bound organelles, such as chloroplast, mitochondria, endoplasmic reticulum, and Golgi apparatus which together differentiates it from a prokaryotic cell
Discovery In 1928 it was discovered by Frederick Griffith in an experiment generally known as transformation.
Experiment
In his experiment he considered two strains of <em>streptococus pneumonia,</em> one was R-type which was non-virulent and cause no disease in mice, other was virulent and S-type which cause disease and at last death of mice.
This experiment was comprised of four steps which are as follow:
Step 1: First he injected living strain of S into mice, after sometime mouce died.
Step 2: He injected living strain of R into mice, the mice alive as he did not got any disease.
Step 3: He injected heat killed strain of S into mice and mice remain alive.
Step 4: He mixed living R strain with heat killed S strain and then inject into mice. As a result the mice died.
Conclusion: It was found that genetic material from heat killed S stain were transferred to living R (non-virulent) strain, as a result R become virulent and cause the death of mice.
Answer:
A. Molecular changes such as extracellular signals on extracellular ligands can result in quick changes in cell behaviour. One example is insulin. Upon insulin binding on the receptor on the cell membrane, the cell release GLUT4 transporters (for muscle cells) to increase uptake of glucose.
B. Slow changes in cell behaviour can be observed for other lipophilic hormones or intracellular receptors such as glucocoortoid or estrogen which reacts with receptors in the cells.
C. Insulin is required to maintain a constant blood glucose level and hence levels of insulin has to be mediated in correspondence to the blood glucose levels. Fast acting signalling is thus required for homeostasis of blood glucose levels. Where for such lipophilic hormones such as steriods, glucocortoid, these hormones tend to have a longer lasting effect and hence results in a slow change.