Answer:
The answer is A a loss of surfactant.
Explanation:
The surfactant is a liquid that intervenes in the capillary-alveolus interface favoring the exchange of gases (oxygen, CO2) at this pulmonary level. For this reason, damage to type II pneumocytes will produce an alteration of this exchange.
Answer:
The missing options are:
a. "It's due to insufficient production of vitamin B12 in the colon."
b. "Increased production of intrinsic factor in the stomach leads to this type of anemia."
c. "Overproduction of vitamin B12 in the large intestine can result in pernicious anemia."
d. "Decreased production of intrinsic factor by the stomach affects the absorption of vitamin B12 in the small intestine."
The correct answer is d. "Decreased production of intrinsic factor by the stomach affects the absorption of vitamin B12 in the small intestine."
Explanation:
Intrinsic factor is a protein that is produced by cells in the stomach, this protein is essential for the absorption of vitamin B12 in the small intestine. Without intrinsic factor, the absorption of vitamin B12 would be really low, causing pernicious anemia since vitamin B12 is necessary for the production of healthy red blood cells. If there is not a good production of healthy red blood cells that can distribute O₂ and CO₂, the body suffers from pernicious anemia.
Answer:
A. Inhibit FSH secretion.
Explanation:
Inhibin is a hormone secreted by the granulosa cells in the ovaries of women and the Sertolli cells in men. The main function of inhibin, as they name says, is to inhibit the secretion of FSH by the anterior pituitary gland. Inhibin is also produced by the Sertolli cells in the testes of men, and it is released in the blood when the sperm count is too high. FSH causes the Sertoli cells of the testes to begin the process of spermatogenesis in the testes. Therefore, releasing inhibin would cause a negative feedback and stop the production of sperm.
