Answer:
If he inherited a mutation which made him more susceptible to lung cancer, it may have been present in some of the gametes he produced and passed to his children
Explanation:
Even tho the cause of lung cancer is not very clear, a genetic predisposition is of a great influence, his smoking and therefore causing a lung cancer is not appliable to his children because of no connection, but in the sense of having a mutation which makes you predisposable to the cancer with or without the smoking, can lead to a high risk of gene inheritance and therefore inheriting the mutation with a high risk of getting lung cancer excluding the smoking.
The plants that were allowed to self pollinate were the F1 plants.
The plants that are true breeding are P generation plants.
The plants where there were 3times as many tall plants as short plants are in F2 generation.
<h3><u>Explanation:</u></h3>
This question is based on the Mendel’s Experiment. Sir Gregor Johann Mendel was the father of genetics who experimented on garden pea plants <em>Pisum</em> <em>sativum</em> to see whether the characters got mixed or not and to know the real cause behind different traits of same character in plants.
He took the pure homozygous tall and short plants separately which he called as parental generation or P generation. These plants were homozygous, hence pure breeding.
As these plants were crossed between themselves, then the F1 generation showed all tall plants. This is because of the heterozygous plants which showed character of dominant trait. These plants were allowed to self pollinate.
As a result of self pollination of the F1 plants, the F2 plants were 75% tall in number whereas the other 25% short, which gave the phenotypic ratio of 3:1.
Mitochondrion because it’s the powerhouse of the cell.
