DNA splitting is DNA replication.
Answer:
Thanks for you question. Your hypothesis suggests a linear relationship between serum Cholesterol levels and MI. This hypothesis seems to ignore the difference in the prevalence and effectiveness of LDL receptors in the FH patient.
FH patients who have inherited the mutation from both parents have very few LDL receptors in their blood and therefore almost no ability to pass the unused Cholesterol through the liver. FH patients who are heterozygous will have more LDL receptors although both will find Cholesterol removal problematic without the addition of a PCSK9 inhibitor.
In short, your hypothesis need to account for other factors that are in play.
Explanation:
Consider my case. I am a 64 year old male who has Heterozygous Familial Hypercholesterolemia. Before treatment at age 12 my Total cholesterol was 510 mg/dl. My genetic testing shows two mutations to the LDL Receptor gene with only one mutation being pathogenic. My first heart attack was at 47 and first stroke at 62. My current LDL is too low to detect with the use of a PCSK9 inhibitor (Repatha®).
Answer:
Organisms compete for shelter
Explanation:
Just took the test and answered this
Plants:
1. Most plants respond to the environment by growing towards the light.
2. Plants respond to daily and seasonal cycles and diseases.
Flatworms:
1. Flatworms respond to changes in light.
2. Flatworms adapt to marine life by using diffusion to breathe and to distribute nutrients to other parts of the body.
Humans:
1. The nervous system allows an organism to respond quickly to changes in the internal or external environment.
2. Some humans modify the environment (positively or negatively), such as cutting forests, building dams, and expanding urban areas, etc.
The endocrine system "regulates" the reproductive system.