Specific chemicals are bound by carrier proteins and transferred on one side of the membrane. The conformational changes they go through next enable the molecule to cross the membrane and exit on the other side.
How carrier protein facilitate the diffusion?
When a molecule diffuses, it usually moves from a high concentration location to a low concentration area until the concentration is the same everywhere in the space.
Contrary to channel proteins, another form of membrane transport protein that is less selective in the molecules it transports, carriers are proteins that move a particular material through intracellular compartments, into the extracellular fluid, or across cells. Carrier proteins are found in lipid bilayer cell structures such cell membranes, mitochondria, and chloroplasts, just like other membrane transport proteins.
Therefore, carrier proteins can facilitate the diffusion of glucose or other substances into the cell.
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Dna damage can occur as a result of exposure to chemicals or ultraviolet radiation. This is what happens during nucleotide excision repair of damaged dna. A multienzyme complex crosses the DNA in search of distortions in the double helix, once located, the phosphate skeleton of the affected chain is cut on both sides of the altered region, a helicase eliminates the oligonucleotide resulting from digestion, the "Gap" "is filled with a DNA polymerase and a ligase.
Answer:
A rare disease that causes damage to the cell's ribosomes will have as a direct effect the inability to perform protein synthesis.
Explanation:
Ribosomes are cellular organelles formed by proteins and a type of RNA called ribosomal RNA, whose function is to translate the genetic code of the mRNA codon sequence and convert it into amino acids. This implies the first step for protein synthesis to occur.
<em><u>If a rare disease could structurally or functionally affect the ribosomes, the translation of mRNA into amino acids could not occur, so protein synthesis in the cell would not be possible</u></em>.
The consequences of the lack of protein synthesis involve severe consequences on the structure and function of a living organism.
The correct answer is the last statement.
If the regulatory serine is mutated to alanine, then acetyl-CoA carboxylase will get activated spontaneously and will produce malonyl-CoA. The increased concentrations of malonyl-CoA will obstruct the oxidation of fatty acids by preventing the entry of fatty acids into the mitochondria.
It is because the AMP-activated protein kinase phosphorylates the serine residues of acetyl-CoA carboxylase to inactivate it. If a mutation occurs in such residues, then the AMPL cannot phosphorylate acetyl-CoA carboxylase and this enzyme will get activated spontaneously.
In such a situation, there will be more than sufficient production of malonyl-CoA, which will inhibit the admittance of more fatty acid getting inside the mitochondria; this will indirectly prevent the oxidation of fatty acids.
Answer:
Blood pressure measurements
Explanation:
Dependent variables are the values that are measurable.
